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Chronic maxillary atelectasis (CMA) is a rare acquired condition of persistent and progressive reduction in maxillary sinus volume and antral wall collapse secondary to ostiomeatal obstruction and development of negative intra-sinus pressure gradients.A 32-year old male was referred with a 6 week history of persistent and worsening sinonasal symptoms, following a significant upper respiratory tract infection. Imaging confirmed bilateral stage I CMA and successful treatment entailed bilateral endoscopic uncinectomy and maxillary antrostomy.Review of the literature has demonstrated CMA to describe an all-encompassing disease process of ostiomeatal obstruction and atelectatic maxillary sinus remodelling that overcomes early variations in taxonomy ('silent sinus syndrome', 'imploding antrum syndrome', 'acquired maxillary sinus hypoplasia') and inconsistencies in reporting. Unilateral CMA is well documented, however a systematic search of the literature reveals only six bilateral cases. To the best of our knowledge, this is the first individual report of bilateral stage I CMA in which the inciting event is established and a uniquely rapid progress of disease followed.The present literature regarding CMA is incomplete and further investigation is required to provide greater insight into its aetiology and pathogenesis. Minimally invasive endoscopic approaches can be employed to re-establish aeration to the affected maxillary sinus for symptomatic relief, to halt disease progression and facilitate antral remodelling and sinus re-expansion.

作者:Dakshika A, Gunaratne;Zubair, Hasan;Peter, Floros;Narinder, Singh

来源:International journal of surgery case reports 2016 年 26卷

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作者:
Dakshika A, Gunaratne;Zubair, Hasan;Peter, Floros;Narinder, Singh
来源:
International journal of surgery case reports 2016 年 26卷
标签:
Chronic maxillary atelectasis Endoscopic sinus surgery Maxillary sinus Sinusitis
Chronic maxillary atelectasis (CMA) is a rare acquired condition of persistent and progressive reduction in maxillary sinus volume and antral wall collapse secondary to ostiomeatal obstruction and development of negative intra-sinus pressure gradients.A 32-year old male was referred with a 6 week history of persistent and worsening sinonasal symptoms, following a significant upper respiratory tract infection. Imaging confirmed bilateral stage I CMA and successful treatment entailed bilateral endoscopic uncinectomy and maxillary antrostomy.Review of the literature has demonstrated CMA to describe an all-encompassing disease process of ostiomeatal obstruction and atelectatic maxillary sinus remodelling that overcomes early variations in taxonomy ('silent sinus syndrome', 'imploding antrum syndrome', 'acquired maxillary sinus hypoplasia') and inconsistencies in reporting. Unilateral CMA is well documented, however a systematic search of the literature reveals only six bilateral cases. To the best of our knowledge, this is the first individual report of bilateral stage I CMA in which the inciting event is established and a uniquely rapid progress of disease followed.The present literature regarding CMA is incomplete and further investigation is required to provide greater insight into its aetiology and pathogenesis. Minimally invasive endoscopic approaches can be employed to re-establish aeration to the affected maxillary sinus for symptomatic relief, to halt disease progression and facilitate antral remodelling and sinus re-expansion.